Acute myeloid leukemia : methods and protocols /

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Bibliographic Details
Group Author: Fortina, Paolo (Editor); Londin, Eric (Editor); Park, Jason Y. (Editor); Kricka, Larry J., 1947- (Editor)
Published: Humana Press,
Publisher Address: New York, NY :
Publication Dates: [2017]
Literature type: eBook
Language: English
Series: Methods in molecular biology, 1633
Springer protocols
Subjects:
Carrier Form: xi, 278 pages : illustrations (some color), forms ; 26 cm.
Bibliography: Includes bibliographical references and index.
ISBN: 9781493971404 (hardback : alkaline paper) :
1493971409 (hardback : alkaline paper)
9781493971428 (eBook)
1493971425 (eBook)
Index Number: RC643
CLC: R733.71
Call Number: R733.71/A189-1
Contents: Molecular malfeasance mediating myeloid malignancies : the genetics of acute myeloid leukemia /
Cytochemical staining /
Immunohistochemistry in acute myeloid leukemia /
Immunophenotyping by multiparameter flow cytometry /
Single-cell mass cytometry of acute myeloid leukemia and leukemia stem/progenitor cells /
Design and application of multiplex PCR seq for the detection of somatic mutations associated with myeloid malignancies /
AMLprofiler : a diagnostic and prognostic microarray for acute myeloid leukemia /
Microsphere-based assessment of DNA methylation for AML prognosis /
Genome-wide analysis of DNA methylation in hematopoietic cells : DNA methylation analysis by WGBS /
Detection and quantification of acute myeloid leukemia-associated fusion transcripts /
Targeted next-generation sequencing of acute leukemia /
Bioinformatics data analysis of next-generation sequencing data from heterogeneous tumor samples /
Zebrafish model for evaluating the function of human leukemic gene IDH1 and its mutation /
Measurement of oncometabolites d-2-hydroxyglutaric acid and l-2-hydroxyglutaric acid /
Nanofluidic allele-specific digital PCR method for quantifying IDH1 and IDH2 mutation burden in acute myeloid leukemia /
Isolation of biologically active exosomes from plasma of patients with cancer /
Generating and expanding autologous chimeric antigen receptor T cells from patients with acute myeloid leukemia /