Atherosclerosis molecular and cellular mechanisms /

Covering the most devastating disease in the Western world, this handbook and ready reference provides a comprehensive account of the different stages and factors in the development of the atherosclerotic plaque. Each chapter is written by experts in the field and highlights the role of specific med...

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Bibliographic Details
Corporate Authors: Wiley InterScience (Online service)
Group Author: George, Sarah J.; Johnson, Jason, Dr.
Published:
Literature type: Electronic eBook
Language: English
Subjects:
Online Access: http://onlinelibrary.wiley.com/book/10.1002/9783527629589
Summary: Covering the most devastating disease in the Western world, this handbook and ready reference provides a comprehensive account of the different stages and factors in the development of the atherosclerotic plaque. Each chapter is written by experts in the field and highlights the role of specific mediators of atherosclerotic plaque development, as well as potential therapeutic targets. A large amount of this up-to-date information is conveyed by way of tables and schematic figures, in a readily comprehensible manner. A must-have for master and PhD students, researchers, MDs and lecturers in vas.
Carrier Form: 1 online resource (xxi, 398 pages) : illustrations
Bibliography: Includes bibliographical references and index.
ISBN: 9783527629589
3527629580
9783527629596 (electronic bk.)
3527629599 (electronic bk.)
Index Number: RC692
CLC: R541.4
Contents: Atherosclerosis: Molecular and Cellular Mechanisms; Contents; Preface; List of Contributors; PART I: Introduction; 1: Pathogenesis of Atherosclerosis; PART II: PRO-Inflammatory Factors; 2: Chemokines and Atherosclerosis: A Critical Assessment of Therapeutic Targets; 3: Adhesion Molecules and Atherosclerosis; 4: Cytokines and Atherosclerosis; 5: Toll-Like Receptors in Atherosclerosis; 6: PPAR-Based Therapies for the Management of Atherosclerosis; 7: Pentraxins in Vascular Pathology: The Role of PTX3; PART III: Proteases; 8: Metalloproteinases, the Endothelium, and Atherosclerosis.