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Advances in cancer research. Volume 141, Cancer stem cells /

Cancer Stem Cells, Volume 141 in the Advances in Cancer Research series, presents the latest release in this ongoing, well-regarded serial that provides invaluable information on the exciting and fast-moving field of cancer research. Topics covered in this new release include SIX-EYA-DACH network co...

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Bibliographic Details
Group Author: Civin, Curt I. (Editor); Kingsbury, Tami J. (Editor); Kim, Minjung (Editor); Fisher, Paul B (Editor)
Published: Academic Press,
Publisher Address: Cambridge, MA :
Publication Dates: 2019.
Literature type: Book
Language: English
Edition: First edition.
Series: Advances in cancer research, volume 141
Subjects:
Cancer cells > Growth > Regulation.
Cancer cells > Differentiation.
Stem cells.
Summary: Cancer Stem Cells, Volume 141 in the Advances in Cancer Research series, presents the latest release in this ongoing, well-regarded serial that provides invaluable information on the exciting and fast-moving field of cancer research. Topics covered in this new release include SIX-EYA-DACH network control of cancer stem cell properties, Dormancy and the cancer cell niche, Clonal hematopoiesis: A hematopoietic stem cell disorder of aging, Stringent assays to study human breast cancer stem cells, Regulation of breast cancer stem cell specification and maintenance by hypoxia-inducible factors, Cancer stem cells in breast and prostate: fact or fiction, and much more.
Carrier Form: xiii, 253 pages : illustrations (some color) ; 24 cm.
Bibliography: Includes bibliographical references.
ISBN: 9780128149942
0128149949
Index Number: RC268
CLC: R73-1
Call Number: R73-1/A244/v.141
Contents: Front Cover; Cancer Stem Cells; Copyright; Contents; Contributors; Preface; References; Chapter One: Regulation of cancer stem cell properties by SIX1, a member of the PAX-SIX-EYA-DACH network; 1. Introduction; 2. Cancer stem cells (CSCs); 3. SIX1 and CSCs; 4. Sustained proliferative signaling; 5. Evading growth suppressors; 6. Enabling replicative immortality; 7. Resisting cell death; 8. Genome instability and mutation; 9. Deregulating cellular energetics; 10. Inducing angiogenesis; 11. Regulation of SIX1 expression; 12. SIX1 clinical significance; 13. EYA; 14. DACH; 15. Concluding remarks.

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